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Article
Open AccessPrevalence of polymorphisms of the genes responsible for auto-inflammatory diseases among 236 patients with recurrent fever in a rheumatology institute in Japan
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Article
Open AccessPReS-FINAL-2125: A Japanese girl with childhood-onset anti-Ku antibody positive generalized morphea-myositis overlap syndrome
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Open AccessPReS-FINAL-2108: Long-term outcome of 114 adult JIA patients in a non-pediatric rheumatology institute in Japan
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Open AccessPW02-024 - A case of candle syndrome treated with thalidomide
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Article
Open AccessEfficacy of thalidomide for a girl with inflammatory calicinosis, a severe complication of juvenile dermatomyosistis
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Article
Open AccessFever without apparent sources in children: a nation-wide study in Japan
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Article
Open Access2.1 Long-term safety and efficacy of tocilizumab in children with systemic juvenile idiopathic arthritis (JIA)
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Article
IL-6 as a therapeutic target in systemic-onset juvenile idiopathic arthritis
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Article
Split chimerism after allogeneic bone marrow transplantation in Chediak–Higashi syndrome
Chediak–Higashi Syndrome (CHS) is a hereditary multiorgan disease associated with a lymphoproliferative disorder termed ‘accelerated phase’ (AP). As AP is often life-threatening, hematopoietic stem cell transp...
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DOPA is a Causal Factor for Glutamate Release and Delayed Neuron Death by Transient Ischemia in Striatum and Hippocampal CA1 of Conscious Rats
DOPA is believed to be an inert amino acid that affects Parkinson’s disease via conversion to dopamine (DA) by aromatic L-amino acid decarboxylase (AADC). We proposed that DOPA is a transmitter and/or modulato...
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Chapter
Involvement of Ionotropic Glutamate Receptors and Nitric Oxide in DOPA-Induced Depressor Responses in the Nucleus Tractus Solitarii
L-Dihydroxyphenylalanine (DOPA) is proposed to be a neurotransmitter for the primary baroreceptor afferents terminating in the nucleus tractus solitarii (NTS),1 and it produces glutamate-like depressor responses ...