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  1. Article

    Open Access

    Prevalence of polymorphisms of the genes responsible for auto-inflammatory diseases among 236 patients with recurrent fever in a rheumatology institute in Japan

    T Miyamae, M Kawamoto, Y Kawaguchi, H Yamanaka in Pediatric Rheumatology (2015)

  2. Article

    Open Access

    PReS-FINAL-2125: A Japanese girl with childhood-onset anti-Ku antibody positive generalized morphea-myositis overlap syndrome

    T Kishi, T Miyamae, A Seki, M Shioda, K Ishigaki, R Morimoto in Pediatric Rheumatology (2013)

  3. Article

    Open Access

    PReS-FINAL-2108: Long-term outcome of 114 adult JIA patients in a non-pediatric rheumatology institute in Japan

    T Miyamae, E Tanaka, T Kishi, T Matsuyama, T Igarashi, S Fujikawa in Pediatric Rheumatology (2013)

  4. Article

    Open Access

    PW02-024 - A case of candle syndrome treated with thalidomide

    K Yamazaki, T Miyamae, S Yokota in Pediatric Rheumatology (2013)

  5. Article

    Open Access

    Efficacy of thalidomide for a girl with inflammatory calicinosis, a severe complication of juvenile dermatomyosistis

    T Miyamae, M Kikuchi, K Kasai, R Hara, U Kaneko, T Shinoki in Pediatric Rheumatology (2008)

  6. Article

    Open Access

    Fever without apparent sources in children: a nation-wide study in Japan

    K Kasai, M Mori, R Hara, T Miyamae, T Imagawa, S Yokota in Pediatric Rheumatology (2008)

  7. Article

    Open Access

    2.1 Long-term safety and efficacy of tocilizumab in children with systemic juvenile idiopathic arthritis (JIA)

    S Yokota, T Imagawa, T Miyamae, K Kasai, M Mori, N Nishimoto in Pediatric Rheumatology (2008)

  8. Article

    IL-6 as a therapeutic target in systemic-onset juvenile idiopathic arthritis

    S Yokota, T Miyamae, T Imagawa, M Mori in Arthritis Res Ther (2003)

  9. No Access

    Article

    Split chimerism after allogeneic bone marrow transplantation in Chediak–Higashi syndrome

    Chediak–Higashi Syndrome (CHS) is a hereditary multiorgan disease associated with a lymphoproliferative disorder termed ‘accelerated phase’ (AP). As AP is often life-threatening, hematopoietic stem cell transp...

    S Yamazaki, H Takahashi, H Fujii, T Miyamae, M Mori in Bone Marrow Transplantation (2003)

  10. No Access

    Chapter

    DOPA is a Causal Factor for Glutamate Release and Delayed Neuron Death by Transient Ischemia in Striatum and Hippocampal CA1 of Conscious Rats

    DOPA is believed to be an inert amino acid that affects Parkinson’s disease via conversion to dopamine (DA) by aromatic L-amino acid decarboxylase (AADC). We proposed that DOPA is a transmitter and/or modulato...

    Y. Misu, Y. Goshima, N. Furukawa, N. Arai, T. Miyamae in Catecholamine Research (2002)

  11. No Access

    Chapter

    Involvement of Ionotropic Glutamate Receptors and Nitric Oxide in DOPA-Induced Depressor Responses in the Nucleus Tractus Solitarii

    L-Dihydroxyphenylalanine (DOPA) is proposed to be a neurotransmitter for the primary baroreceptor afferents terminating in the nucleus tractus solitarii (NTS),1 and it produces glutamate-like depressor responses ...

    Y. Goshima, K. Yamanashi, T. Miyamae, Y. Sasaki, M. Maeda in Catecholamine Research (2002)