Skip to main content

2,915 Result(s)

within Neurosurgery Remove this filter **u-**u Zhang Remove this filter Medicine & Public Health Remove this filter

Page of 146
and

  1. No Access

    Chapter and Conference Paper

    Indomethacin and Cyclosporin a Inhibit in Vitro Ischemia-Induced Expression of ICAM-1 and Chemokines in Human Brain Endothelial Cells

    Brain inflammation has been implicated in the development of brain edema and secondary brain damage in ischemia and trauma. Mechanisms involved in leukocyte infiltration across the blood-brain barrier are stil...

    W. Zhang, C. Smith, R. Monette, J. Hutchison, Dr. D. B. Stanimirovic in Brain Edema XI (2000)

  2. No Access

    Chapter and Conference Paper

    Thrombosis After Ischemic Stroke-Platelet Aggregation

    After ischemic stroke secondary thrombosis contributes to the expansion of the lesion. This manuscript describes the underlying mechanisms responsible for cerebral thrombosis after stroke. We hypothesize that ...

    M. Chopp, Z. G. Zhang in Maturation Phenomenon in Cerebral Ischemia IV (2001)

  3. No Access

    Chapter and Conference Paper

    Delayed Clot Removal and Experimental Vasospasm

    It is known that vasospastic arteries have reduced compliance and contractility and exhibit changes in expression of various proteins [3, 1]. It is possible that they may be altered in a manner that reduces their...

    Z.-D. Zhang, B. Yamini, T. Komuro, S. Ono, L. Johns, L. S. Marton… in Cerebral Vasospasm (2001)

  4. No Access

    Chapter and Conference Paper

    A Novel Drug Delivery System as Prophylaxis for Cerebral Vasospasm

    Experimental work has extensively demonstrated that calcium antagonists can reverse established angiographically identified vasospasm when administered by the intrathecal route, despite being ineffective when ...

    Y. Takanashi M.D., Ph.D., T. Ishida, T. Meguro, M. J. Kirchmeier… in Cerebral Vasospasm (2001)

  5. No Access

    Chapter and Conference Paper

    Apoptosis in Cerebral Vasospasm After Subarachnoid Hemorrhage

    We hypothesize that the mechanism of cerebral vasospasm after subarachnoid hemorrhage may not be a delayed and prolonged vascular response to certain spasmogens but a more complicated cascade, including contra...

    Kotaro Ogihara, Alexander Y. Zubkov, Robert E. Tibbs… in Ischemic Blood Flow in the Brain (2001)

  6. No Access

    Chapter and Conference Paper

    Molecular Weight Interactions in Experimental Vasospasm

    Vasospasm is believed to be primarily due to the release of substances from the subarachnoid clot that act upon the arterial wall to contract the artery initially and then produce pharmacological and morpholog...

    R. Loch Macdonald M.D., Ph.D., S. Ono, L. Johns, L. S. Marton… in Cerebral Vasospasm (2001)

  7. No Access

    Chapter and Conference Paper

    Complimentary Deoxyribonucleic Acid Array Analysis of Cerebral Arteries after Experimental Subarachnoid Hemorrhage

    Cerebral vasospasm is a prolonged vasoconstriction that develops on average 3 days after subarachnoid hemorrhage (SAH), peaks 7 to 8 days after the hemorrhage and then slowly resolves over the next 5 to 7 days [6

    Z.-D. Zhang, L. S. Marton, S. Ono, T Komuro, B. Weir… in Cerebral Vasospasm (2001)

  8. No Access

    Chapter and Conference Paper

    Apoptosis in Basilar Endothelial Cells in a Canine Double Hemorrhage Model

    Cerebral vasospasm after subarachnoid hemorrhage (SAH) is a combination of prolonged contraction and vessel remodeling that may lead to cerebral ischemia or infarction [2]. Morphological studies of cerebral arter...

    A. Y. Zubkov, R. E. Tibbs, B. Clower, K. Ogihara, K. Aoki… in Cerebral Vasospasm (2001)

  9. No Access

    Chapter and Conference Paper

    Role of MAPK in Chronic Cerebral Vasospasm

    Many extracellular stimuli (including growth factors, hormones, stress, elevated temperature and G-protein coupled receptor agonists) initialize the phosphorylation cascades leading to the activation of mitoge...

    K. Aoki, A. Y. Zubkov, R. E. Tibbs, T. Meguro… in Cerebral Vasospasm (2001)

  10. No Access

    Chapter and Conference Paper

    Role of Mitogen-Activated Protein Kinase in the Contractions of Rabbit Basilar Artery Induced by Hemolysate, Oxyhemoglobin, and Bloody CSF

    It has been suggested that tyrosine kinases are involved in smooth muscle contraction [1] induced by hemolysate or other spasmogens and might be involved in cerebral vasospasm. Mitogen-activated protein kinase (M...

    A. Y. Zubkov, K. Shadon Rollins, B. McGehee, A. D. Parent… in Cerebral Vasospasm (2001)

  11. No Access

    Chapter

    Cell Signaling: Serine/Threonine Protein Kinases and Traumatic Brain Injury

    Traumatic brain injury (TBI) is a complex interaction of primary and secondary processes that may require days to evolve. Injury processes are related to the magnitude and type of TBI and the resulting systemi...

    Larry Jenkins, C. Edward Dixon, Grant Peters, Wei-Min Gao, **aopeng Zhang… in Brain Injury (2001)

  12. No Access

    Chapter

    Apoptosis

    Apoptosis, or programmed cell death, is a complex and strictly regulated cellular event. A prerequisite for apoptosis is the existence of highly conserved death programs within the cell genome. The induction o...

    **aopeng Zhang, Margaret A. Satchell, Robert S. B. Clark… in Brain Injury (2001)

  13. No Access

    Article

    Primary malignant lymphoma of the brain: analysis of MMAC1 (PTEN) tumor suppressor gene

    With the use of RT-PCR (reverse transcriptase-polymerase chain reaction), Northern blot analysis, and Western blot analysis, seven primary brain lymphomas were examined for the state of the MMAC1 tumor suppres...

    **n Ding, Sumio Endo, Shu-**g Zhang, Takafumi Saito, Mituo Kouno… in Brain Tumor Pathology (2001)

  14. No Access

    Chapter and Conference Paper

    Metabolic Alterations in Cerebrospinal Fluid from Double Hemorrhage Model of Dogs

    Even though cerebral vasospasm after subarachnoid hemorrhage (SAH) causes cerebral ischemia or infarction, the metabolic alterations in cerebrospinal fluids (CSF) after SAH have not been studied. This study wa...

    W. Yin, R. Tibbs, K. Aoki, A. Badr… in Intracranial Pressure and Brain Biochemica… (2002)

  15. No Access

    Article

    Regional differences within the human supraspinous and interspinous ligaments: a sheet plastination study

    The extent to which neighboring muscles and the fascia contribute to the formation of the supraspinous and interspinous ligaments is not clear from the literature. The purpose of this investigation is to exam...

    Gillian M. Johnson, Ming Zhang in European Spine Journal (2002)

  16. Article

    Open Access

    Chromosome 4q;10q translocations; Comparison with different ethnic populations and FSHD patients

    Facioscapulohumeral muscular dystrophy (FSHD) is an autosomal dominant disorder characterized by the weakness of facial, shoulder-girdle and upper arm muscles. Most patients with FSHD have fewer numbers of tan...

    Tsuyoshi Matsumura, Kanako Goto, Gaku Yamanaka, Je Hyeon Lee, Cheng Zhang… in BMC Neurology (2002)

    Download PDF (839 KB) View Article

  17. No Access

    Chapter and Conference Paper

    Multiple hyperbaric oxygenation (HBO) expands the therapeutic window in acute spinal cord injury in rats

    Hyperbaric oxygenation (HBO) therapy has been reported to improve neurological recovery following spinal cord injury (SCI). In the present study, we examined whether multiple HBO expands the therapeutic window...

    L. Huang, M. P. Mehta, J. H. Eichhorn, A. Nanda… in Brain Edema XII (2003)

  18. No Access

    Chapter and Conference Paper

    Highly polysialylated neural cell adhesion molecule (PSA-NCAM) positive cells are increased and change localization in rat hippocampus by exposure to repeated kindled seizures

    The highly polysialylated neural cell adhesion molecule (PSANCAM) is involved in migration of neural stem cells as well as in neural plasticity. Immunoreactive PSA-NCAM expression was examined in rats with rep...

    K. Sato M.D., M. Iwai, W.-R. Zhang, H. Kamada, K. Ohta, N. Omori… in Brain Edema XII (2003)

  19. No Access

    Chapter and Conference Paper

    Matrix metalloproteinases are not involved in early brain edema formation after cardiac arrest in rats

    Resuscitation from cardiac arrest (CA) often results in a poor neurological outcome possibly due to an incomplete understanding of the pathophysiology of brain injury following CA-induced global cerebral ische...

    Feng **ao M.D., T. Arnold, S. Zhang, N. Imtiaz, A. Khan, J. S. Alexander… in Brain Edema XII (2003)

  20. No Access

    Chapter and Conference Paper

    Heat shock proteins expression in brain stem after subarachnoid hemorrhage in rats

    The pathogenesis of brain damage after subarachnoid hemorrhage (SAH) especially at molecular or gene level remains unclear. We used complimentary deoxyribonucleic acid (cDNA) macroarray technique and compared ...

    M. Satoh, J. Tang, A. Nanda, John H. Zhang M.D., Ph.D. in Brain Edema XII (2003)

Page of 146