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    Article

    James Watson Kernohan (1896–1981)

    D. E. Haines, J. A. Lancon, A. Y. Zubkov in Journal of Neurology (2005)

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    Chapter and Conference Paper

    Apoptosis in Basilar Endothelial Cells in a Canine Double Hemorrhage Model

    Cerebral vasospasm after subarachnoid hemorrhage (SAH) is a combination of prolonged contraction and vessel remodeling that may lead to cerebral ischemia or infarction [2]. Morphological studies of cerebral arter...

    A. Y. Zubkov, R. E. Tibbs, B. Clower, K. Ogihara, K. Aoki in Cerebral Vasospasm (2001)

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    Chapter and Conference Paper

    Role of MAPK in Chronic Cerebral Vasospasm

    Many extracellular stimuli (including growth factors, hormones, stress, elevated temperature and G-protein coupled receptor agonists) initialize the phosphorylation cascades leading to the activation of mitoge...

    K. Aoki, A. Y. Zubkov, R. E. Tibbs, T. Meguro in Cerebral Vasospasm (2001)

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    Chapter and Conference Paper

    Role of Mitogen-Activated Protein Kinase in the Contractions of Rabbit Basilar Artery Induced by Hemolysate, Oxyhemoglobin, and Bloody CSF

    It has been suggested that tyrosine kinases are involved in smooth muscle contraction [1] induced by hemolysate or other spasmogens and might be involved in cerebral vasospasm. Mitogen-activated protein kinase (M...

    A. Y. Zubkov, K. Shadon Rollins, B. McGehee, A. D. Parent in Cerebral Vasospasm (2001)

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    Chapter and Conference Paper

    Mitogen-Activated Protein Kinase Plays an Important Role in Hemolysate-Induced Contraction in Rabbit Basilar Artery

    Object. Mitogen-activated protein kinase (MAPK) is an important signaling factor in the vascular proliferation and contraction, the two features of cerebral vasospasm following subarachnoid hemor...

    A. Y. Zubkov, K. Ogihara, A. Patllola, A. D. Parent in Brain Edema XI (2000)

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    Chapter and Conference Paper

    Oxyhemoglobin Produces Necrosis in Cultured Smooth Muscle Cells

    Object. Myonecrosis in the tunica media, which is defined morphologically, is one of the most striking alterations in the cerebral arterial wall following subarachnoid hemorrhage (SAH). In this s...

    K. Ogihara, A. Y. Zubkov, A. D. Parent, John H. Zhang M.D., Ph.D. in Brain Edema XI (2000)