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Article
Role and therapeutic potential of dietary ketone bodies in lymph vessel growth
Lymphatic vessels (LVs), lined by lymphatic endothelial cells (LECs), are indispensable for life1. However, the role of metabolism in LECs has been incompletely elucidated. In the present study, it is reported th...
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Article
The role of fatty acid β-oxidation in lymphangiogenesis
Lymphatic vessels are lined by lymphatic endothelial cells (LECs), and are critical for health. However, the role of metabolism in lymphatic development has not yet been elucidated. Here we report that in tran...
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Article
Open AccessGlycolytic regulation of cell rearrangement in angiogenesis
During vessel sprouting, endothelial cells (ECs) dynamically rearrange positions in the sprout to compete for the tip position. We recently identified a key role for the glycolytic activator PFKFB3 in vessel s...
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Article
Correction: Corrigendum: PAI-1 mediates the antiangiogenic and profibrinolytic effects of 16K prolactin
Nat. Med. 20, 741–747 (2014); published online 15 June 2014; corrected after print 25 August 2014 In the version of this article initially published, it was stated that 16K PRL levels are increased in retinopa...
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Article
Open AccessMyocyte enhancer factor 2C function in skeletal muscle is required for normal growth and glucose metabolism in mice
Skeletal muscle is the most abundant tissue in the body and is a major source of total energy expenditure in mammals. Skeletal muscle consists of fast and slow fiber types, which differ in their energy usage, ...
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Article
Correction: Corrigendum: PAI-1 mediates the antiangiogenic and profibrinolytic effects of 16K prolactin
Nat. Med. 20, 741–747 (2014); published online 15 June 2014; corrected after print 25 August 2014 In the version of this article initially published, it was stated that 16K PRL levels are increased in retinopa...
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Article
PAI-1 mediates the antiangiogenic and profibrinolytic effects of 16K prolactin
The antiangiogenic and profibrinolytic activities of the N-terminal fragment of prolactin, 16K PRL, are mediated by its binding to the protein PAI-1.
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Article
Impaired myocardial angiogenesis and ischemic cardiomyopathy in mice lacking the vascular endothelial growth factor isoforms VEGF164 and VEGF188