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    MicroRNA-17-3p suppresses NF-κB-mediated endothelial inflammation by targeting NIK and IKKβ binding protein

    Nuclear factor kappa B (NF-κB) activation contributes to many vascular inflammatory diseases. The present study tested the hypothesis that microRNA-17-3p (miR-17-3p) suppresses the pro-inflammatory responses v...

    Yin Cai, Yu Zhang, Hui Chen, **ng-hui Sun, Peng Zhang in Acta Pharmacologica Sinica (2021)

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    IL-17A promotes ventricular remodeling after myocardial infarction

    Inflammatory responses play an important role in the pathogenesis of adverse ventricular remodeling after myocardial infarction (MI). We previously demonstrated that interleukin (IL)-17A plays a pathogenic rol...

    Su-Feng Zhou, **g Yuan, Meng-Yang Liao, Ni **a in Journal of Molecular Medicine (2014)

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    Regulatory T cells ameliorate cardiac remodeling after myocardial infarction

    Persistent inflammatory responses participate in the pathogenesis of adverse ventricular remodeling after myocardial infarction (MI). We hypothesized that regulatory T (Treg) cells modulate inflammatory respon...

    Ting-Ting Tang, **g Yuan, Zheng-Feng Zhu, Wen-Cai Zhang in Basic Research in Cardiology (2011)

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    Poly (ADP-ribose) Polymerase Inhibition Improves Endothelial Dysfunction Induced by Hyperhomocysteinemia in Rats

    We investigated the possible protective effect of poly (ADP-ribose) polymerase (PARP) inhibition in preventing endothelial dysfunction induced by hyperhomocysteinemia (Hhcy).

    **an Yu, **ang Cheng, Jiang-jiao **e, Meng-yang Liao in Cardiovascular Drugs and Therapy (2009)