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Article
Open AccessCD147 a direct target of miR-146a supports energy metabolism and promotes tumor growth in ALK+ ALCL
We recently reported that miR-146a is differentially expressed in ALK+ and ALK− anaplastic large cell lymphoma (ALCL). In this study, the downstream targets of miR-146a in ALK+ ALCL were investigated by transc...
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Article
Open AccessSuper-enhancer-based identification of a BATF3/IL-2R−module reveals vulnerabilities in anaplastic large cell lymphoma
Anaplastic large cell lymphoma (ALCL), an aggressive CD30-positive T-cell lymphoma, comprises systemic anaplastic lymphoma kinase (ALK)-positive, and ALK-negative, primary cutaneous and breast implant-associat...
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Article
Open AccessSTAT3 and TP53 mutations associate with poor prognosis in anaplastic large cell lymphoma
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Article
Open AccessCorrection: Dependency on the TYK2/STAT1/MCL1 axis in anaplastic large cell lymphoma
An amendment to this paper has been published and can be accessed via a link at the top of the paper.
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Article
Effect of Iron-Aluminide Coating on the Fracture Mechanism of Ferritic–Martensitic Steel in Coal-Fired Boilers Environment
Due to the coal combustion that generates halides, steel components can confront hot corrosion during applications at high temperature. The hot-dip** aluminum (HDA) was operated on the 9Cr–Mo steel (grade 91...
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Article
Open AccessDependency on the TYK2/STAT1/MCL1 axis in anaplastic large cell lymphoma
TYK2 is a member of the JAK family of tyrosine kinases that is involved in chromosomal translocation-induced fusion proteins found in anaplastic large cell lymphomas (ALCL) that lack rearrangements activating ...
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Article
Open AccessThe AP-1-BATF and -BATF3 module is essential for growth, survival and TH17/ILC3 skewing of anaplastic large cell lymphoma
Transcription factor AP-1 is constitutively activated and IRF4 drives growth and survival in ALK+ and ALK– anaplastic large cell lymphoma (ALCL). Here we demonstrate high-level BATF and BATF3 expression in ALCL. ...
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Article
Erratum to: The collective nuclear migration of p53 and phosphorylated S473 of Akt during ellipticine-mediated apoptosis in human lung epithelial cancer cells
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Article
The collective nuclear migration of p53 and phosphorylated S473 of Akt during ellipticine-mediated apoptosis in human lung epithelial cancer cells
Topoisomerase II inhibitor ellipticine effectively suppressed the growth of human non-small-cell-lung-cancer (NSCLC) epithelial cells. Previously, we reported the drug activity was consummated through paralle...
