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  1. No Access

    Chapter and Conference Paper

    Cerebroprotective Effects of a Parenteral Flunarizine Formulation

    Since the synthesis of flunarizine over 25 years ago, there have been over 230 experimental and 150 clinical peer-reviewed descriptions of its pharmacological activity. According to the proposed WHO classifica...

    H. L. Edmonds Jr., G. Raque, P. Y. Zhang, S. A. Jenkins… in Cerebral Ischemia and Calcium (1989)

  2. No Access

    Chapter and Conference Paper

    Calcium Movements in Brain During Failure of Energy Metabolism

    The interstitial ion concentrations in brain are maintained within narrow limits. Changes in plasma concentrations are effectively dealt with by the low ionic permeability of the blood-brain barrier combined w...

    E. Zhang, M. Lauritzen, T. Wieloch, A. J. Hansen in Cerebral Ischemia and Calcium (1989)

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    Article

    Hormone receptors in vestibular schwannomas

    The possibility that steroid hormones play a role in vestibular schwannoma proliferation has been suggested by a number of investigators. There is conflicting information about the presence of steroid hormone ...

    R. S. Carroll, J. P. Zhang, P. Mc L. Black in Acta Neurochirurgica (1997)

  4. No Access

    Article

    Osteosarcoma of the meninges in association with glioblastoma

    Primary intracranial osteosarcoma not originating in the skull is a distinctly rare tumour, as is post-irradiation sarcoma of short latency. The authors report the case of a 56 year old caucasian male who unde...

    W. T. Couldwell M.D., Ph.D., B. W. Scheithauer, S. G. Rice… in Acta Neurochirurgica (1997)

  5. No Access

    Article

    Apoptosis in astrocytic neoplasms

    Apoptosis is a form of programmed cell death characterized by specific morphologic and biochemical properties. Tumorgenesis is the consequence not only of cell proliferation but also the loss of the ability to...

    R. S. Carroll, J. Zhang, B. W. Chauncey, K. Chantziara… in Acta Neurochirurgica (1997)

  6. No Access

    Chapter and Conference Paper

    The Clot Thickens — Thrombolysis and Combination Therapies

    Stroke in the human is most frequently caused by an intra-arterial clot. In order to investigate human stroke, appropriate and relevant animal models must be selected. Since the only approved treatment of stro...

    Michael Chopp Ph.D., R. L. Zhang… in Current Progress in the Understanding of S… (1999)

  7. No Access

    Chapter and Conference Paper

    Delayed Gene Expression and Ischemic Brain Injury

    There is increasing evidence that cerebral ischemic injury occurs at a slower pace than previously believed. Although in areas of severe ischemia tissue damage occurs relatively rapidly, in regions of less-sev...

    C. Iadecola, M. E. Ross, F. Zhang, S. Nogawa… in Maturation Phenomenon in Cerebral Ischemia… (1999)

  8. No Access

    Chapter and Conference Paper

    Protein Expression and Brain Plasticity After Transient Middle Cerebral Artery Occlusion in the Rat

    After a stroke, both animals and humans can regain lost function. The molecular basis of this functional plasticity is unknown. In this manuscript, we describe developmental protein expression in the ischemic ...

    M. Chopp, Y. Li, Z. G. Zhang in Maturation Phenomenon in Cerebral Ischemia III (1999)

  9. No Access

    Chapter and Conference Paper

    Morphological Presentation of Posttraumatic Vasospasm

    Posttraumatic vasospasm is a well-recognized sequela of head injury. The risk factors associated with posttraumatic vasospasm have not been well defined. We studied 119 consecutive patients with head injury to...

    Alexander Y. Zubkov M.D., A. S. Pilkington, A. D. Parent, J. Zhang in Brain Edema XI (2000)

  10. No Access

    Chapter and Conference Paper

    Mitogen-Activated Protein Kinase Plays an Important Role in Hemolysate-Induced Contraction in Rabbit Basilar Artery

    Object. Mitogen-activated protein kinase (MAPK) is an important signaling factor in the vascular proliferation and contraction, the two features of cerebral vasospasm following subarachnoid hemor...

    A. Y. Zubkov, K. Ogihara, A. Patllola, A. D. Parent… in Brain Edema XI (2000)

  11. No Access

    Chapter and Conference Paper

    Role of Tyrosine Kinase in Fibroblast Compaction and Cerebral Vasospasm

    Hemolysate, a proposed causative agent for cerebral vasospasm following subarachnoid hemorrhage, produces contraction of cerebral arteries by activation of tyrosine kinases. In addition, hemolysate accelerates...

    A. Patlolla, K. Ogihara, A. Zubkov, K. Aoki, A. D. Parent… in Brain Edema XI (2000)

  12. No Access

    Chapter and Conference Paper

    Oxyhemoglobin Produces Necrosis in Cultured Smooth Muscle Cells

    Object. Myonecrosis in the tunica media, which is defined morphologically, is one of the most striking alterations in the cerebral arterial wall following subarachnoid hemorrhage (SAH). In this s...

    K. Ogihara, A. Y. Zubkov, A. D. Parent, John H. Zhang M.D., Ph.D. in Brain Edema XI (2000)

  13. No Access

    Chapter and Conference Paper

    Indomethacin and Cyclosporin a Inhibit in Vitro Ischemia-Induced Expression of ICAM-1 and Chemokines in Human Brain Endothelial Cells

    Brain inflammation has been implicated in the development of brain edema and secondary brain damage in ischemia and trauma. Mechanisms involved in leukocyte infiltration across the blood-brain barrier are stil...

    W. Zhang, C. Smith, R. Monette, J. Hutchison, Dr. D. B. Stanimirovic in Brain Edema XI (2000)

  14. No Access

    Chapter and Conference Paper

    Thrombosis After Ischemic Stroke-Platelet Aggregation

    After ischemic stroke secondary thrombosis contributes to the expansion of the lesion. This manuscript describes the underlying mechanisms responsible for cerebral thrombosis after stroke. We hypothesize that ...

    M. Chopp, Z. G. Zhang in Maturation Phenomenon in Cerebral Ischemia IV (2001)

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    Chapter and Conference Paper

    Delayed Clot Removal and Experimental Vasospasm

    It is known that vasospastic arteries have reduced compliance and contractility and exhibit changes in expression of various proteins [3, 1]. It is possible that they may be altered in a manner that reduces their...

    Z.-D. Zhang, B. Yamini, T. Komuro, S. Ono, L. Johns, L. S. Marton… in Cerebral Vasospasm (2001)

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    Chapter and Conference Paper

    A Novel Drug Delivery System as Prophylaxis for Cerebral Vasospasm

    Experimental work has extensively demonstrated that calcium antagonists can reverse established angiographically identified vasospasm when administered by the intrathecal route, despite being ineffective when ...

    Y. Takanashi M.D., Ph.D., T. Ishida, T. Meguro, M. J. Kirchmeier… in Cerebral Vasospasm (2001)

  17. No Access

    Chapter and Conference Paper

    Apoptosis in Cerebral Vasospasm After Subarachnoid Hemorrhage

    We hypothesize that the mechanism of cerebral vasospasm after subarachnoid hemorrhage may not be a delayed and prolonged vascular response to certain spasmogens but a more complicated cascade, including contra...

    Kotaro Ogihara, Alexander Y. Zubkov, Robert E. Tibbs… in Ischemic Blood Flow in the Brain (2001)

  18. No Access

    Chapter and Conference Paper

    Molecular Weight Interactions in Experimental Vasospasm

    Vasospasm is believed to be primarily due to the release of substances from the subarachnoid clot that act upon the arterial wall to contract the artery initially and then produce pharmacological and morpholog...

    R. Loch Macdonald M.D., Ph.D., S. Ono, L. Johns, L. S. Marton… in Cerebral Vasospasm (2001)

  19. No Access

    Chapter and Conference Paper

    Complimentary Deoxyribonucleic Acid Array Analysis of Cerebral Arteries after Experimental Subarachnoid Hemorrhage

    Cerebral vasospasm is a prolonged vasoconstriction that develops on average 3 days after subarachnoid hemorrhage (SAH), peaks 7 to 8 days after the hemorrhage and then slowly resolves over the next 5 to 7 days [6

    Z.-D. Zhang, L. S. Marton, S. Ono, T Komuro, B. Weir… in Cerebral Vasospasm (2001)

  20. No Access

    Chapter and Conference Paper

    Apoptosis in Basilar Endothelial Cells in a Canine Double Hemorrhage Model

    Cerebral vasospasm after subarachnoid hemorrhage (SAH) is a combination of prolonged contraction and vessel remodeling that may lead to cerebral ischemia or infarction [2]. Morphological studies of cerebral arter...

    A. Y. Zubkov, R. E. Tibbs, B. Clower, K. Ogihara, K. Aoki… in Cerebral Vasospasm (2001)

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