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  1. No Access

    Article

    DEK promotes HPV-positive and -negative head and neck cancer cell proliferation

    Head and neck squamous cell carcinoma (HNSCC) is the sixth most common malignancy worldwide, and patient outcomes using current treatments remain poor. Tumor development is etiologically associated with tobacc...

    A K Adams, G E Hallenbeck, K A Casper, Y J Patil, K M Wilson, R J Kimple… in Oncogene (2015)

  2. No Access

    Article

    High incidence of female reproductive tract cancers in FA-deficient HPV16-transgenic mice correlates with E7’s induction of DNA damage response, an activity mediated by E7’s inactivation of pocket proteins

    Fanconi anemia (FA) is a rare genetic disorder caused by defects in a DNA damage repair system, the FA pathway. FA patients frequently develop squamous cell carcinoma (SCC) at sites that are associated with hu...

    J W Park, M-K Shin, P F Lambert in Oncogene (2014)

  3. No Access

    Article

    The immortalizing and transforming ability of two common human papillomavirus 16 E6 variants with different prevalences in cervical cancer

    Persistent infection with high-risk human papillomaviruses (HPVs), especially type 16 has been undeniably linked to cervical cancer. The Asian-American (AA) variant of HPV16 is more common in the Americas than...

    C Richard, C Lanner, S N Naryzhny, L Sherman, H Lee, P F Lambert, I Zehbe in Oncogene (2010)

  4. No Access

    Article

    HPV16 E6 confers p53-dependent and p53-independent phenotypes in the epidermis of mice deficient for E6AP

    High-risk human papillomaviruses are the causative agents of cervical and other anogenital cancers. In these cancers, two viral oncogenes, E6 and E7, are expressed. E6 is best known for its ability to inactiva...

    A Shai, M L Nguyen, J Wagstaff, Y-h Jiang, P F Lambert in Oncogene (2007)