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  1. Article

    Genetic variants affecting incretin sensitivity and incretin secretion

    Recent genome-wide association studies identified several novel risk genes for type 2 diabetes. The majority of these type 2 diabetes risk variants confer impaired pancreatic beta cell function. Though the mol...

    K. Müssig, H. Staiger, F. Machicao, H.-U. Häring, A. Fritsche in Diabetologia (2010)

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  2. Article

    The ENPP1 K121Q polymorphism determines individual susceptibility to the insulin-sensitising effect of lifestyle intervention

    The K121Q (rs1044498) single nucleotide polymorphism (SNP) in the ENPP1 gene has shown association with insulin resistance and type 2 diabetes in various ethnic populations. We hypothesised that K121Q may predict...

    K. Müssig, M. Heni, C. Thamer, K. Kantartzis, F. Machicao, N. Stefan… in Diabetologia (2010)

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  3. Article

    A common genetic variant in WFS1 determines impaired glucagon-like peptide-1-induced insulin secretion

    WFS1 type 2 diabetes risk variants appear to be associated with impaired beta cell function, although it is unclear whether insulin secretion is affected directly or secondarily via alteration ...

    S. A. Schäfer, K. Müssig, H. Staiger, F. Machicao, N. Stefan, B. Gallwitz… in Diabetologia (2009)

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    Article

    Paraneoplastisches ektopes ACTH-Syndrom als Folge eines kleinzelligen Lungenkarzinoms

    Dr. M. Zachäus, K. Müssig in Der Internist (2008)

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    Article

    Symptomatic hypoglycemia during imatinib mesylate in a non-diabetic female patient with gastrointestinal stromal tumor

    Imatinib mesylate is a selective competitive inhibitor of the bcr-abl tyrosine kinase and c-KIT. Other kinases, such as phosphatidylinositol 3′-kinase (PI-3K) involved in insulin signaling, have also been show...

    M. Haap, B. Gallwitz, C. Thamer, K. Müssig… in Journal of Endocrinological Investigation (2007)

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    Article

    Adrenocortical carcinoma producing 11-deoxycorticosterone: A rare cause of mineralocorticoid hypertension

    A 37-yr-old man presented with the classic signs of mineralocorticoid excess hypertension and hypokalemia. The cause was not aldosterone excess, but elevation of plasma 11-deoxycorticosterone (DOC). Computed t...

    K. Müssig, M. Wehrmann, M. Horger… in Journal of Endocrinological Investigation (2005)

  7. Article

    40th EASD Annual Meeting of the European Association for the Study of Diabetes

    M. Veitenhansl, K. Stegner, F.-X. Hierl, C. Dieterle, H. Feldmeier, B. Gutt… in Diabetologia (2004)

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