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  1. No Access

    Article

    C-terminal BRE overexpression in 11q23-rearranged and t(8;16) acute myeloid leukemia is caused by intragenic transcription initiation

    Overexpression of the BRE (brain and reproductive organ-expressed) gene defines a distinct pediatric and adult acute myeloid leukemia (AML) subgroup. Here we identify a promoter enriched for active chromatin mark...

    A E Marneth, K H M Prange, A S A Al Hinai, S M Bergevoet, N Tesi… in Leukemia (2018)

  2. Article

    Erratum: miR-194-5p/BCLAF1 deregulation in AML tumorigenesis

    Correction to: Leukemia (2017) 31, 2315–2325; doi:10.1038/leu.2017.64; published online 10 March 2017 Following the publication of this article, the authors noted a mistake in the author affiliations. 4. Centr...

    C Dell'Aversana, C Giorgio, L D'Amato, G Lania, F Matarese, S Saeed… in Leukemia (2018)

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  3. Article

    Open Access

    miR-194-5p/BCLAF1 deregulation in AML tumorigenesis

    Deregulation of epigenetic mechanisms, including microRNA, contributes to leukemogenesis and drug resistance by interfering with cancer-specific molecular pathways. Here, we show that the balance between miR-1...

    C Dell'Aversana, C Giorgio, L D'Amato, G Lania, F Matarese, S Saeed… in Leukemia (2017)

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  4. Article

    Open Access

    CBFB–MYH11/RUNX1 together with a compendium of hematopoietic regulators, chromatin modifiers and basal transcription factors occupies self-renewal genes in inv(16) acute myeloid leukemia

    Different mechanisms for CBFβ–MYH11 function in acute myeloid leukemia with inv(16) have been proposed such as tethering of RUNX1 outside the nucleus, interference with transcription factor complex assembly an...

    A Mandoli, A A Singh, P W T C Jansen, A T J Wierenga, H Riahi, G Franci… in Leukemia (2014)

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