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  1. No Access

    Article

    Higher Plasma Osteopontin Concentrations Associated with Subsequent Development of Chronic Shunt-Dependent Hydrocephalus After Aneurysmal Subarachnoid Hemorrhage

    A matricellular protein osteopontin (OPN) is considered to exert neuroprotective and healing effects on neurovascular injuries in an acute phase of aneurysmal subarachnoid hemorrhage (SAH). However, the relati...

    Reona Asada, Yoshinari Nakatsuka, Hideki Kanamaru… in Translational Stroke Research (2021)

  2. No Access

    Chapter

    Possible Involvement of Caspase-Independent Pathway in Neuronal Death After Subarachnoid Hemorrhage in Mice

    Early brain injury is now considered as an important cause of delayed neurological deterioration after aneurysmal subarachnoid hemorrhage (SAH), and neuronal apoptosis is one of the constituents of early brain...

    Fumi Nakano, Lei Liu, Fumihiro Kawakita, Yoshinari Nakatsuka… in Subarachnoid Hemorrhage (2020)

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    Chapter

    Link Between Receptors That Engage in Develo** Vasospasm After Subarachnoid Hemorrhage in Mice

    Vasospasm after subarachnoid hemorrhage (SAH) has been studied, but the mechanisms remain to be unveiled. Tenascin-C (TNC), which is a matricellular protein and reported to increase in spastic cerebral artery ...

    Fumi Nakano, Fumihiro Kawakita, Lei Liu, Yoshinari Nakatsuka… in Subarachnoid Hemorrhage (2020)

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    Chapter

    The Role of Galectin-3 in Subarachnoid Hemorrhage: A Preliminary Study

    Despite advances in diagnosis and treatment of subarachnoid hemorrhage (SAH), combined morbidity and mortality rate in SAH patients accounted for greater than 50%. Many prognostic factors have been reported in...

    Hirofumi Nishikawa, Fumi Nakano, Lei Liu, Yoshinari Nakatsuka… in Subarachnoid Hemorrhage (2020)

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    Chapter

    TAK-242, Toll-Like Receptor 4 Antagonist, Attenuates Brain Edema in Subarachnoid Hemorrhage Mice

    Background: Brain edema is a common and critical pathology following subarachnoid hemorrhage (SAH). Toll-like receptor 4 (TLR4) activation may exacerbate brain edema. The purpose of this study was to clarify if ...

    Takeshi Okada, Liu Lei, Hirofumi Nishikawa, Fumi Nakano… in Subarachnoid Hemorrhage (2020)

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    Article

    Dose-Dependent Inhibitory Effects of Cilostazol on Delayed Cerebral Infarction After Aneurysmal Subarachnoid Hemorrhage

    Cilostazol is a selective inhibitor of phosphodiesterase type III that downregulates tenascin-C (TNC), a matricellular protein, which may cause delayed cerebral infarction after aneurysmal subarachnoid hemorrh...

    Hidenori Suzuki, Yoshinari Nakatsuka, Ryuta Yasuda… in Translational Stroke Research (2019)

  7. No Access

    Article

    Increased Plasma Galectin-3 Preceding the Development of Delayed Cerebral Infarction and Eventual Poor Outcome in Non-Severe Aneurysmal Subarachnoid Hemorrhage

    A matricellular protein galectin-3 is involved in tissue injury and inflammation, but the role of galectin-3 remains unclear in aneurysmal subarachnoid hemorrhage (SAH). The purpose of this study was to assess...

    Hirofumi Nishikawa, Yoshinari Nakatsuka, Masato Shiba… in Translational Stroke Research (2018)

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    Article

    Higher Cerebrospinal Fluid pH may Contribute to the Development of Delayed Cerebral Ischemia after Aneurysmal Subarachnoid Hemorrhage

    Recent investigations have shown that many factors may cause delayed cerebral ischemia (DCI) after aneurysmal subarachnoid hemorrhage (SAH). To find new potential contributors to DCI, this retrospective study ...

    Hidenori Suzuki, Masato Shiba, Yoshinari Nakatsuka… in Translational Stroke Research (2017)