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Article
Longitudinal study of 8 plasma mineralocorticoids(MC), glucocorticoids(GC) and progestins in premature infants (PI) at birth and during the neonatal period
To evaluate fetoplacental and adrenocortical functions of the PI at birth and during postnatal adaptation, plasma aldosterone(Aldo), corticosterone(B), 11-deoxycorticosterone(DOC), progesterone(P), 17-hydroxyp...
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Article
EVIDENCE FOR A NATURALLY OCCURING MINERALOCORTICOIDANTAGONIST IN CONGENITAL ADRENAL HYPERPLASIA (CAH)
The existence of a salt-excreting factor in CAH due to a 21-hydroxylase defect has been postulated for many years.However extensive investigations sofar were unable to isolate such a factor.Our demonstration o...
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Article
Etomidate (E): a potent inhibitor of adrenocortical 11 β-hydroxylase activity
Increased mortality with long-term E infusions in intensive care patients has been reported and evidence is accumulating that E causes reversible adrenocortical suppression with decreased serum cortisol levels...
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Article
SMALL FOR GESTATIONAL AGE (SGA) INFANTS: PLASMA MINERALOCORTICOIDS, GLUCOCORTICOIDS AND PR0GESTINS AT BIRTH AND DURING THE NEONATAL PERIOD
The small adrenal cortex of SGA infants may play a role in the pathogenesis of their frequent postnatal problems. We therefore evaluated adrenocortical function of SGA infants at birth and during postnatal ada...
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Article
PITFALLS OF PRENATAL TREATMENT OF CONGENITAL ADRENAL HYPERPLASIA (CAH) DUE TO 21-HYDROXYLASE DEFICIENCY
It has been reported that prenatal dexamethasone (DEX) treatment of women carrying female CAH fetuses suppresses fetal adrenal function and prevents virilization of external genitalia. In 2 CAH families, the m...
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Article
PRENATAL TREATMENT OF CONGENITAL ADRENAL HYPERPLASIA (CAH) DUE TO 21-HYDROXYLASE DEFICIENCY: EUROPEAN EXPERIENCE IN 223 PREGNANCIES AT RISK
In the aim to prevent the in utero virilization of CAH affected females a prenatal treatment (Rx) has first been attempted by David and Forest in 1979; later these authors reported their first results and propose...