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Chapter and Conference Paper
Renal Tubular Handling of Urate in Calcium Stone Formers with Hyperuricosuria or Renal Acidification Defects
Hyperuricosuria has been considered as a risk factor for calcium stone formation (1) and thought to be mainly a result of consumption of purine rich animal protein (2). According to Coe (3) some hyperuricosuri...
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Chapter
Long-term Treatment with Bendroflumethiazide for Prevention of Stones. Metabolic Effects
Since 1975 we have used thiazides for prevention of renal calcium stones. Our experience1 confirms that of Yendt and Cohanim2 that in most cases stone formation will virtually cease during several years of follow...
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Chapter
Prophylactic Treatment with Magnesium Hydroxide in Renal Stone Disease
Hammarsten1 demonstrated 50 years ago that magnesium could increase the solubility of calcium oxalate in vitro and proposed that treatment with magnesium could be used to prevent renal calcium stones. Others2–3 h...
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Chapter
Magnesium Metabolism in Renal Stone Formers
A possible role for magnesium in the formation of calcium stones was suggested by the investigations of Hammarsten1, who reported a beneficial effect of magnesium on the solubility of calcium oxalate in vitro. Fu...
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Chapter
Experience with the Short Ammonium Chloride Test
Since renal tubular acidosis is often complicated by stone formation, it seemed justified to investigate a stone forming population in order to study the incidence of renal acidification defects. For this purp...
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Chapter
Urate Metabolism in Calcium Stone Disease
Urate metabolism has been extensively investigated in calcium stone disease. Some investigators have reported that hyperuricemia was a common feature among stone formers1; whereas this could not be reproduced by ...
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Chapter
Beta-2-Microglobulin Excretion in Renal Stone-Formers
Renal tubular defects in patients with recurring kidneystones have not been paid much attention to, except in patients with RTA. In order to investigate the proximal tubular function in renal stone formers the...