3 Result(s)
within
Stephan Theiss
Cancer Research
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Article
Open AccessBalanced splicing at the Tat-specific HIV-1 3′ss A3 is critical for HIV-1 replication
The viral regulatory protein Tat is essential for establishing a productive transcription from the 5′-LTR promoter during the early phase of viral gene expression. Formation of the Tat-encoding mRNAs requires ...
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Article
Open AccessHEXploring of the HIV-1 genome allows landsca** of new potential splicing regulatory elements
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Article
Comparative in silico analyses and experimental validation of novel splice site and missense mutations in the genes MLH1 and MSH2
Hereditary non-polyposis colorectal cancer, an autosomal dominant predisposition to colorectal cancer and other malignancies, is caused by inactivating mutations of DNA mismatch repair genes, mainly MLH1 and MSH2
