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  1. Article

    Erratum: VAV1 regulates experimental autoimmune arthritis and is associated with anti-CCP negative rheumatoid arthritis

    Correction to: Genes and Immunity (2017) 18, 48–56; doi:10.1038/gene.2016.49; published online 5 January 2017 Since the publication of this article the authors found that there were errors in titles of Tables ...

    A O Guerreiro-Cacais, U Norin, A Gyllenberg, R Berglund, A D Beyeen in Genes & Immunity (2017)

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    Article

    VAV1 regulates experimental autoimmune arthritis and is associated with anti-CCP negative rheumatoid arthritis

    Rheumatoid arthritis (RA) patients can be stratified into two subgroups defined by the presence or absence of antibodies against citrullinated circular peptides (anti-CCP) with most of the genetic association ...

    A O Guerreiro-Cacais, U Norin, A Gyllenberg, R Berglund, A D Beyeen in Genes & Immunity (2017)

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    Article

    Finemap** of the arthritis QTL Pia7 reveals co-localization with Oia2 and the APLEC locus

    In this study, we sought to determine the effect of the quantitative trait locus Pia7 on arthritis severity. The regulatory locus derived from the arthritis-resistant E3 rat strain was introgressed into the arthr...

    C Rintisch, T Kelkka, U Norin, J C Lorentzen, P Olofsson, R Holmdahl in Genes & Immunity (2010)

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    Article

    Gene expression profiling of arthritis using a QTL chip reveals a complex gene regulation of the Cia5 region in mice

    One of the major quantitative trait loci (QTLs) associated with arthritis in crosses between B10.RIII and RIIIS/J mice is the Cia5 on chromosome 3. Early in the congenic map** process it was clear that the locu...

    M Johannesson, L M Olsson, A-K B Lindqvist, S Möller, D Koczan in Genes & Immunity (2005)

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    Article

    Identification of epistasis through a partial advanced intercross reveals three arthritis loci within the Cia5 QTL in mice

    Identification of genes controlling complex diseases has proven to be difficult; however, animal models may pave the way to determine how low penetrant genes interact to promote disease development. We have di...

    M Johannesson, J Karlsson, P Wernhoff, K S Nandakumar, A-K Lindqvist in Genes & Immunity (2005)