![Loading...](https://link.springer.com/static/c4a417b97a76cc2980e3c25e2271af3129e08bbe/images/pdf-preview/spacer.gif)
-
Article
Erratum: VAV1 regulates experimental autoimmune arthritis and is associated with anti-CCP negative rheumatoid arthritis
Correction to: Genes and Immunity (2017) 18, 48–56; doi:10.1038/gene.2016.49; published online 5 January 2017 Since the publication of this article the authors found that there were errors in titles of Tables ...
-
Article
VAV1 regulates experimental autoimmune arthritis and is associated with anti-CCP negative rheumatoid arthritis
Rheumatoid arthritis (RA) patients can be stratified into two subgroups defined by the presence or absence of antibodies against citrullinated circular peptides (anti-CCP) with most of the genetic association ...
-
Article
The multiple sclerosis risk gene IL22RA2 contributes to a more severe murine autoimmune neuroinflammation
Single-nucleotide polymorphisms close to IL22RA2, coding for the soluble interleukin (IL)-22-binding protein (IL-22BP), are strongly and reproducibly associated with multiple sclerosis (MS), but there is little d...
-
Article
Anti-MOG antibodies are under polygenic regulation with the most significant control coming from the C-type lectin-like gene locus
Multiple sclerosis (MS) is a chronic inflammatory disease of the central nervous system that is genetically complex. There is evidence supporting a role of myelin oligodendrocyte glycoprotein (MOG) humoral imm...