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  1. No Access

    Chapter and Conference Paper

    Protective effect of the V1a receptor antagonist SR49059 on brain edema formation following middle cerebral artery occlusion in the rat

    There exists no pharmacological treatment for fulminating brain edema. Since evidence indicates that brain aquaporin-4 (AQP4) water channels are modulated by vasopressin V1a receptors, we examined the edema-re...

    A. Kleindienst, G. Fazzina, J. G. Dunbar, R. Glisson, A. Marmarou in Brain Edema XIII (2006)

  2. No Access

    Chapter and Conference Paper

    Bolus tracer delivery measured by MRI confirms edema without blood-brain barrier permeability in diffuse traumatic brain injury

    Previous studies have shown that edema formation after diffuse traumatic brain injury (TBI) with secondary insult is cytotoxic and not vasogenic. This assumption is based on observations of reduced apparent di...

    A. Beaumont, P. Fatouros, T. Gennarelli, F. Corwin, A. Marmarou in Brain Edema XIII (2006)

  3. No Access

    Chapter and Conference Paper

    Modulation of AQP4 expression by the protein kinase C activator, phorbol myristate acetate, decreases ischemia-induced brain edema

    The protein kinase C activator, phorbol 12-myristate 13-acetate (PMA), is known to interact with aquaporin-4 (AQP4), a waterselective transporting protein abundant in astrocytes and ependymal cells, that has b...

    A. Kleindienst, G. Fazzina, A. M. Amorini, J. G. Dunbar, R. Glisson… in Brain Edema XIII (2006)

  4. No Access

    Chapter and Conference Paper

    Traumatic brain edema in diffuse and focal injury: cellular or vasogenic?

    The objective of this study was to confirm the nature of the edema, cellular or vasogenic, in traumatic brain injury in head-injured patients using magnetic resonance imaging techniques. Diffusion-weighted ima...

    A. Marmarou, S. Signoretti, G. Aygok, P. Fatouros, G. Portella in Brain Edema XIII (2006)

  5. No Access

    Chapter and Conference Paper

    Brain tissue water content in patients with idiopathic normal pressure hydrocephalus

    Relatively little is known regarding the water content of brain tissue in idiopathic normal-pressure hydrocephalus (NPH) patients. The objective of our study was to determine absolute water content non-invasiv...

    G. Aygok, A. Marmarou, P. Fatouros, H. Young in Brain Edema XIII (2006)

  6. No Access

    Chapter and Conference Paper

    Effect of dimethyl sulfoxide on blood-brain barrier integrity following middle cerebral artery occlusion in the rat

    Dimethyl sulfoxide (DMSO) is widely used as a solvent for other drugs, i.e., for the protein kinase C activator phorbol 12-myristate 13-acetate (PMA) and the V1a receptor-antagonist SR49059, to reduce brain ed...

    A. Kleindienst, J. G. Dunbar, R. Glisson, K. Okuno, A. Marmarou in Brain Edema XIII (2006)

  7. No Access

    Chapter and Conference Paper

    Comparison of NAA Measures by MRS and HPLC

    This work investigates the accuracy of an in vivo estimation of absolute N-acetyl aspartate (NAA) concentrations by magnetic resonance spectroscopy (MRS) using cerebral water as an internal reference standard....

    P. P. Fatouros Ph.D., D. L. Heath, A. Beaumont, F. D. Corwin… in Brain Edema XI (2000)

  8. No Access

    Chapter and Conference Paper

    CSF and ECF Glutamate Concentrations in Head Injured Patients

    Excitatory Amino Acids (EAAs) release has been considered to be neurotoxic in traumatic brain injury patients. Microdialysis samples of extracellular space (ECS) and high glutamate concentrations in cerebrospi...

    T. Yamamoto, S. Rossi, M. Stiefel, E. Doppenberg… in Neuromonitoring in Brain Injury (1999)

  9. No Access

    Chapter and Conference Paper

    Biphasic Pathophysiological Response of Vasogenic and Cellular Edema in Traumatic Brain Swelling

    The objective of this study was to quantify the temporal water content changes and document the type of edema (cellular versus vasogenic) that is occurring during both the acute and the late stages of edema de...

    P. Barzó, A. Marmarou, P. Fatouros, K. Hayasaki, F. Corwin in Brain Edema X (1997)

  10. No Access

    Chapter and Conference Paper

    The Fine Structural Study of Brain Response to Intracerebral Infusion of Serum in the Cat

    The mechanism of vasogenic edema resolution is unclear and is a subject of investigation at various laboratories, including our own. Our previous studies using the infusion model of brain edema indicated disti...

    A. Hirano, A. Marmarou, T. Nakamura, A. Inoue in Brain Edema (1985)

  11. No Access

    Chapter and Conference Paper

    Development of Brain Edema Following Fluid Percussion Injury

    The progressive increase in brain volume following severe mechanical injury ultimately leads to intracranial ischemia and hypertension. The severity of the problem is in part reflected in the statistic that 50...

    A. Marmarou, T. Nakamura, H. Sakamoto, L. Jenkins, D. P. Becker in Brain Edema (1985)