Erratum to: Flt3 receptor inhibition reduces constitutive NFκB activation in high-risk myelodysplastic syndrome and acute myeloid leukemia

Erratum to: Apoptosis (2008) 13:1148–1161 DOI 10.1007/s10495-008-0243-4

During the preparation of Fig. 3A for the final version of this manuscript, the authors have assembled immunoblots from different cell lines without clearly indicating the borders between blots. A corrected version of Fig. 3A in which borders between blots are clearly indicated, with the loading controls for all the membranes subjected to immunoblot analyses is provided below. The results of the corrected Fig. 3A do not differ from the original version.

Fig. 3

Knockdown of Flt3 causes NFκB inhibition and apoptosis. (a–c) Effect of a pool of Flt3-specific siRNAs. MOLM-13 and MV4-11 cells were electroporated with the indicated siRNA pool, and the expression level and phosphorylation status of Flt3, IKK1/2 and degradation of IκBα were detected by immunoblot 48 h later (a). NFκB activation was measured by EMSA (b) and the frequency of dying cells (X ± SD, n = 3) was determined by DiOC₆(3)/PI staining (c). (d–e) Effect of individual Flt3-specific siRNAs. MOLM-13 and MV4-11 cells were electroporated with 2 distinct Flt3 siRNA and the expression level Flt3 was detected by immunoblot 48 h later (d), NFκB activity was measured by EMSA (f) the frequency of dying cells (X ± SD, n = 3) was determined by DiOC₆(3)/PI staining (e)